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The typical arterial wall is made up of three basic layers. There are also two forms of fat within the body. The fat is called low density lipoprotein (LDL) and high density lipoprotein (HDL). LDL is considered a fat molecule, which can go into the wall and venture deep into the arterial layers.
The mechanism called Atherosclerosis begins with the buildup of LDL accumulates within the arterial wall. This is dependent on the condition of the wall and the concentration of increased LDL levels. LDL will go into the arterial wall and undergoes a mechanism called oxidation. When the LDL is treated to the process of oxidation, it becomes dangerous within the body and triggers the process of inflammation. White blood cells called monocytes become involved and move from the bloodstream into the arterial wall. They start the inflammatory response, turn into cells that are referred to as macrophages, which swallow the oxidized LDL. When this happens they are now called foam cells.
The problem is often that foam cells can burst releasing the contents of the cell. When this happens regularly, the foam cell will form fibrous material. Therefore, continued arteries plaque growth that results from the buildup of LDL in the arterial wall can result in the membrane. This is above and beyond what it can hold.
In the beginning this is a good thing as it allows the walls of the artery to keep a set amount of blood to maintain a noteworthy but reduced the flow. Therefore, tests will show only those arteries plaques that bulge into the center of the arterial lumen. This is because the plaque is located deep within the layers. The results of any test is not the true story of the amount of plaque and Atherosclerosis.
Yet when the amount of plaque continues to grow, the arterial wall is not able to cope with this new development. Therefore, the visible is not able to cope and starts to the process. However, this expanding causes the Atherosclerosis deposit to push into the center of the vessel. This happens when the amount of plaque formed is at least 40 percent. Certain mechanisms can cause the plaque to burst or become dislodged. This is a serious problem especially when the plaque swells and burst or the disruption of the plaque occurs.
Plaques that are unstable will burst, especially those that contain a large amount of lipids covered by a weak fibrous layer. Often these arteries plaque are still located within the arterial wall and not pushed into the arterial lumen. They cannot be easily seen.
If the plaque bursts, the lipid content is brought into contact with blood, which can result in the growth of blood clots. If the vessel is completely blocked for at least two hours, this can cause a serious condition called myocardial infraction, more commonly known as a heart attack.
The mechanism called Atherosclerosis begins with the buildup of LDL accumulates within the arterial wall. This is dependent on the condition of the wall and the concentration of increased LDL levels. LDL will go into the arterial wall and undergoes a mechanism called oxidation. When the LDL is treated to the process of oxidation, it becomes dangerous within the body and triggers the process of inflammation. White blood cells called monocytes become involved and move from the bloodstream into the arterial wall. They start the inflammatory response, turn into cells that are referred to as macrophages, which swallow the oxidized LDL. When this happens they are now called foam cells.
The problem is often that foam cells can burst releasing the contents of the cell. When this happens regularly, the foam cell will form fibrous material. Therefore, continued arteries plaque growth that results from the buildup of LDL in the arterial wall can result in the membrane. This is above and beyond what it can hold.
In the beginning this is a good thing as it allows the walls of the artery to keep a set amount of blood to maintain a noteworthy but reduced the flow. Therefore, tests will show only those arteries plaques that bulge into the center of the arterial lumen. This is because the plaque is located deep within the layers. The results of any test is not the true story of the amount of plaque and Atherosclerosis.
Yet when the amount of plaque continues to grow, the arterial wall is not able to cope with this new development. Therefore, the visible is not able to cope and starts to the process. However, this expanding causes the Atherosclerosis deposit to push into the center of the vessel. This happens when the amount of plaque formed is at least 40 percent. Certain mechanisms can cause the plaque to burst or become dislodged. This is a serious problem especially when the plaque swells and burst or the disruption of the plaque occurs.
Plaques that are unstable will burst, especially those that contain a large amount of lipids covered by a weak fibrous layer. Often these arteries plaque are still located within the arterial wall and not pushed into the arterial lumen. They cannot be easily seen.
If the plaque bursts, the lipid content is brought into contact with blood, which can result in the growth of blood clots. If the vessel is completely blocked for at least two hours, this can cause a serious condition called myocardial infraction, more commonly known as a heart attack.
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If you have been looking for a way to reduce your risk of heart disease, lower your cholesterol, get your blood pressure under control,have a clean arteries in your heart and ultimately Save Your Heart, then the Pauling Therapy is for you! We help how to clean arteries naturally. It is a Heart Treatment That Really Works!
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